The investigator's work highlights how a variation in a person's DNA sequence is associated with an earlier age of onset for breast cancer that is stimulated by the hormone estrogen (estrogen receptor positive). At focus were DNA sequence variations (polymorphisms) in the human enzyme known as HDAC9. Recent studies of this enzyme suggest that its suppression could lead to strategies that would regulate estrogen signaling. This latest research further supports a role for HDAC9 in estrogen signaling in that genetic changes in the HDAC9 gene may modulate onset of ER positive breast cancer and its recurrence.
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